Capt. Clayton Osbon, the JetBlue pilot who experienced a breakdown in midflight last week and had to be subdued by passengers, will stay in jail until a detention hearing on Thursday, according to the?Amarillo Globe-News.?
Osbon was released from the Pavilion at Amarillo's Northwest Texas Healthcare System in handcuffs at 7:30 a.m CT, the paper reported.
Flight 191 was scheduled from John F. Kennedy Airport to Las Vegas, but was diverted to Amarillo, Texas, on Tuesday. JetBlue suspended Osbon last week following the incident. He is charged with interfering with a flight crew and, if convicted, could face 20 years in prison.
Osbon's wife, Connye Osbon, said on Sunday that the pilot's family is focused on his recovery. She asked the media for privacy and said the family would not be granting interviews or making further statements. She said the family appreciated the public's concern.
"It is our belief, as Clayton's family, that while he was clearly distressed, he was not intentionally violent toward anyone," said astatement?by Connye Osbon on behalf of the Osbon family that was released by JetBlue on Sunday. "We know you were placed in an awful situation and we appreciate your ability to respond professionally."
JetBlue said in a?blog post?last week that it will not share further details about the captain's "private life."
Contact: Susan Martonik smartonik@snm.org 703-652-6773 Society of Nuclear Medicine
New data open door to potential diagnostic and treatment options
New research published in the April issue of The Journal of Nuclear Medicine reveals that systemic inflammation causes an increase in depressive symptoms and metabolic changes in the parts of the brain responsible for mood and motivation. With this finding, researchers can begin to test potential treatments for depression for patients that experience symptoms that are related to inflammation in the body or within the brain.
Multiple studies in rodents have shown that inflammation in the body has effects on the brain. This has also been shown in a few human studiesboth through measurements of behavioral changes and brain imagingwhen subjects were engaged in various computer tasks. The study "Glucose Metabolism in the Insula and Cingulate Is Affected by Systemic Inflammation in Humans," however, for the first time measured brain activity when subjects were at rest.
"In the study we used F-18 fluorodeoxyglucose (FDG) positron emission tomography (PET), which can accurately measure glucose metabolism in the brain, to determine which brain regions responded to systemic inflammation. Since the subjects were at rest, the changes we observed in the brain can only attributed to systemic inflammation," noted Jonas Hannestad, MD, PhD, lead author of the article.
In the study, nine healthy individuals received a double-blind endotoxin (which elicits systemic inflammation and mild depressive symptoms such as fatigue and reduced social interest) and placebo on different days. After administration, F-18 FDG PET was used to measure the differences in the cerebral metabolic rate of glucose in the insula, cingulate and amygdala regions of the brain. Behavior changes were also primarily assessed on the Montgomery-Asberg Depression Rating Scale (MADRS).
A statistical analysis of the results showed that endotoxin administration was associated with a higher normalized glucose metabolism (NMG) in the insula and lower NMG in the cingulate compared to the placebo; there was no significant difference in the NMG in the amygdala. Seven of nine subjects had an increase in NMG in the insula and a decrease in NMG in the cingulate, and all nine subjects had a decrease in NMG in the right anterior cingulate, suggesting that systemic inflammation induces fundamental physiologic changes in regional brain glucose metabolism. In addition, the MADRS increased for each subject after endotoxin administration, whereas no significant change was noted with the placebo.
Most researchers agree that depression is not a homogeneous disease, but rather that there are multiple mechanisms that can lead to similar symptoms. "If we can show that a subtype of depression is caused in part by inflammation," said Hannestad, "we can test the ability of treatments that reduce inflammation in only patients in whom we believe inflammation plays a role. In the future, I expect that researchers in this field will be able to develop more precise PET measures that can be used to distinguish between, for instance, a person with 'inflammatory depression' and a person with another kind of depression. PET could then be used as diagnostic biomarker to separate subtypes of depression and as a therapeutic biomarker to detect the response to treatment."
Nearly 17 percent of adults experience depression at some point over their lifetime, with 30.4 percent of cases classified as severe, according to the U.S. National Institute of Mental Health. Fifty-seven percent of adults with depression report receiving treatment in the past 12 months, although 37.8 percent receive minimally adequate treatment.
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Authors of the article "Glucose Metabolism in the Insula and Cingulate Is Affected by Systemic Inflammation in Humans" include: Jonas Hannestad, Kalyani Subramanyam, Nicole DellaGioia and Brian Pittman, Department of Psychiatry, Yale School of Medicine, New Haven, Connecticut; and Beata Planeta-Wilson, David Weinzimmer and Richard E. Carson, Department of Diagnostic Radiology, Yale School of Medicine, New Haven, Connecticut.
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
Contact: Susan Martonik smartonik@snm.org 703-652-6773 Society of Nuclear Medicine
New data open door to potential diagnostic and treatment options
New research published in the April issue of The Journal of Nuclear Medicine reveals that systemic inflammation causes an increase in depressive symptoms and metabolic changes in the parts of the brain responsible for mood and motivation. With this finding, researchers can begin to test potential treatments for depression for patients that experience symptoms that are related to inflammation in the body or within the brain.
Multiple studies in rodents have shown that inflammation in the body has effects on the brain. This has also been shown in a few human studiesboth through measurements of behavioral changes and brain imagingwhen subjects were engaged in various computer tasks. The study "Glucose Metabolism in the Insula and Cingulate Is Affected by Systemic Inflammation in Humans," however, for the first time measured brain activity when subjects were at rest.
"In the study we used F-18 fluorodeoxyglucose (FDG) positron emission tomography (PET), which can accurately measure glucose metabolism in the brain, to determine which brain regions responded to systemic inflammation. Since the subjects were at rest, the changes we observed in the brain can only attributed to systemic inflammation," noted Jonas Hannestad, MD, PhD, lead author of the article.
In the study, nine healthy individuals received a double-blind endotoxin (which elicits systemic inflammation and mild depressive symptoms such as fatigue and reduced social interest) and placebo on different days. After administration, F-18 FDG PET was used to measure the differences in the cerebral metabolic rate of glucose in the insula, cingulate and amygdala regions of the brain. Behavior changes were also primarily assessed on the Montgomery-Asberg Depression Rating Scale (MADRS).
A statistical analysis of the results showed that endotoxin administration was associated with a higher normalized glucose metabolism (NMG) in the insula and lower NMG in the cingulate compared to the placebo; there was no significant difference in the NMG in the amygdala. Seven of nine subjects had an increase in NMG in the insula and a decrease in NMG in the cingulate, and all nine subjects had a decrease in NMG in the right anterior cingulate, suggesting that systemic inflammation induces fundamental physiologic changes in regional brain glucose metabolism. In addition, the MADRS increased for each subject after endotoxin administration, whereas no significant change was noted with the placebo.
Most researchers agree that depression is not a homogeneous disease, but rather that there are multiple mechanisms that can lead to similar symptoms. "If we can show that a subtype of depression is caused in part by inflammation," said Hannestad, "we can test the ability of treatments that reduce inflammation in only patients in whom we believe inflammation plays a role. In the future, I expect that researchers in this field will be able to develop more precise PET measures that can be used to distinguish between, for instance, a person with 'inflammatory depression' and a person with another kind of depression. PET could then be used as diagnostic biomarker to separate subtypes of depression and as a therapeutic biomarker to detect the response to treatment."
Nearly 17 percent of adults experience depression at some point over their lifetime, with 30.4 percent of cases classified as severe, according to the U.S. National Institute of Mental Health. Fifty-seven percent of adults with depression report receiving treatment in the past 12 months, although 37.8 percent receive minimally adequate treatment.
###
Authors of the article "Glucose Metabolism in the Insula and Cingulate Is Affected by Systemic Inflammation in Humans" include: Jonas Hannestad, Kalyani Subramanyam, Nicole DellaGioia and Brian Pittman, Department of Psychiatry, Yale School of Medicine, New Haven, Connecticut; and Beata Planeta-Wilson, David Weinzimmer and Richard E. Carson, Department of Diagnostic Radiology, Yale School of Medicine, New Haven, Connecticut.
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AAAS and EurekAlert! are not responsible for the accuracy of news releases posted to EurekAlert! by contributing institutions or for the use of any information through the EurekAlert! system.
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I don’t know about you, but I’m ready to be implanted with that chip people keep talking about because I want to be always connected.? Until that day, I have to find other ways to keep myself connected.? It seems most every device now offers a cellular option for 3G (or even 4G) connectivity, but [...]
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Yes, of course the president wants his signature health care law upheld by the U.S. Supreme Court... but I suspect (suspect, since no honest person can say he knows) that it would help him politically if the statute were to be struck down in its entirety. This November election could be close and enthusiasm really matters in a close election. So how does he rev up the enthusiasm? Right now he has considerable drag e.g., gas prices.
Striking down the statute could really rev up the president's base -- add fuel to their current political mantra fire about a 'war on women.' I can think of the ads now, can't you? The president's enthusiasm level among his supporters has been sagging -- something that happens during a second term campaign -- and he needs something to throw his weight behind to really drive his supporters to the polls. An existing health care law won't do it since it feels a bit like, 'been there, done it.' Striking the law down, on the other hand, could really be used to put political panic in his supporters.
Plus... if the law is stricken by the U.S. Supreme Court, those current less-than-enthusiastic Republican voters, and the independents furious about health care may not find their way to the polls come November if they have achieved what they already wanted -- getting rid of the health care law. They may feel a bit ho-hum about voting since they got what they wanted.
Of course if some giant international or domestic event happens, health care law will become irrelevant as it gets pushed down our collective radar screen. And of course, much could happen between now and November.
What do you think? Am I wrong? (By the way, people I know who spends lots of time thinking about politics say I am wrong about this one... )
Cross-posted from GretaWire, where it first appeared.
March 31, 2012 (TopWireNews.com: - Business News, Current News, press release)
03/26/2012 // Highlands Ranch, Colorado, United States // FrankieCervantes // The Great Online
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